new paper from pathologists in Kanazawa City, Japan reports the findings of an autopsy in a healthy 40-year-old man:

A healthy 40-year-old Japanese man suddenly experienced tachycardia and lost consciousness two days after vaccination. Continued resuscitation recovered the spontaneous heartbeat; however, the patient did not regain consciousness and died nine days later.

Electrocardiography after resuscitation showed marked ST-segment depression and incomplete right bundle branch block.

Influenza antigen and polymerase chain reaction tests for SARS-CoV-2 were negative.

The key findings at autopsy were (emphasis mine):

At autopsy, several small inflammatory foci with cardiomyocytic necrosis were scattered in the right and left ventricles, with a propensity for the right side.

Some inflammatory foci were located near the atrioventricular nodes and His bundles. The infiltrating cells predominantly consisted of CD68-positive histiocytes, with a small number of CD8-positive and CD4-positive T cells.

In this case, myocarditis was focal and mild, as is mostly observed following COVID-19 mRNA vaccination. However, the inflammatory foci were close to the conduction system and were considered the cause of fatal arrhythmia.

The most concerning aspect of this is that the myocarditis itself was “focal and mild, as is mostly observed following COVID-19 mRNA vaccination”. Yet this resulted in a fatal arrhythmia, because cells which are part of the electrical system were affected.

A secondary concern about this is that since this occurred after the second dose, it seems likely the event happened several years ago, yet has remained unpublished in the meantime.

The possibility that inflammation affecting just the conductive system might result in fatal arrhythmia is a concern that we at the Health Advisory and Recovery Team (HART) have previously raised in relation to the mRNA products.

It is to be noted that this man presented suddenly with an arrhythmia (ventricular tachycardia) which ultimately proved fatal; preceding chest pain characteristic of myocarditis was not reported. This is consistent with our concern that subclinical myocarditis, about which we have written extensively, does indeed result in an increased risk of fatal arrhythmias, which may be responsible for most or all of the rise in ambulance callouts for cardiac arrests, which we have also written about. Subclinical myocarditis – defined as a rise in troponin indicative of some cardiac damage – has been found in at least one in 50 people injected with the mRNA Covid injection in those studies where researchers have routinely looked for it.